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Aims & Scope


DNA and Cell Biology delivers authoritative, peer-reviewed research on all aspects of molecular and cellular biology, with a unique focus on combining mechanistic and clinical studies to drive the field forward.

DNA and Cell Biology coverage includes:

  • Gene Structure, Function, and Regulation
    • Gene regulation
    • Molecular mechanisms of cell activation
    • Mechanisms of transcriptional, translational, or epigenetic control of gene expression
  • Molecular Medicine
    • Molecular pathogenesis
    • Genetic approaches to cancer and autoimmune diseases
    • Translational studies in cell and molecular biology
  • Cellular Organelles
    • Autophagy
    • Apoptosis
    • P bodies
    • Peroxisosomes
  • Protein Biosynthesis and Degradation
    • Regulation of protein synthesis
    • Post-translational modifications
    • Control of degradation
  • Cell-Autonomous Inflammation and Host Cell Response to Infection
    • Responses to cytokines and other physiological mediators
    • Evasive pathways of pathogens

DNA and Cell Biology is under the editorial leadership of Editor-in-Chief Carol Shoshkes Reiss, PhD, Departments of Biology and Neural Science, New York University, and other leading investigators. View the entire editorial board.

Audience: DNA researchers, biomedical researchers, cell biologists, and immunologists, among others

Indexing/Abstracting:

  • PubMed/MEDLINE
  • PubMed Central
  • Scopus
  • Current Contents®/Life Sciences
  • Biotechnology Citation Index®
  • Biological Abstracts
  • BIOSIS Citation Index™
  • Journal Citation Reports/Science Edition
  • EMBASE/Excerpta Medica
  • EMBiology
  • Chemical Abstracts
  • ProQuest databases
  • CAB Abstracts
  • Global Health
  • BenchSci
The views, opinions, findings, conclusions and recommendations set forth in any Journal article are solely those of the authors of those articles and do not necessarily reflect the views, policy or position of the Journal, its Publisher, its editorial staff or any affiliated Societies and should not be attributed to any of them.